Cut-leaved coneflower
General poisoning notes:
Cut-leaved
coneflower (Rudbeckia laciniata) is native to some parts of Canada and
is naturalized in others. A double-flowered form is also used as an ornamental
in flower beds and is usually called golden glow. Early circumstantial evidence
of poisoning of horses, sheep, and swine can be found. Experiments on sheep and
swine have shown that some symptoms of toxicity can occur, although animals
generally refuse to eat the unpalatable plants. Animal poisoning by this plant
should be considered unlikely (Kingsbury 1964).
Nomenclature:
Scientific Name: Rudbeckia laciniata L.
Vernacular
name(s): cut-leaved coneflower
Scientific family
name: Compositae
Vernacular family
name: composite
Geographic Information
Alberta, British
Columbia, Manitoba, New Brunswick, Nova Scotia, Ontario, Prince Edward Island,
Quebec, Saskatchewan
Notes on Poisonous plant parts:
Ingesting the
aboveground portion of this plant produced symptoms in some animals (Kingsbury
1964).
Toxic parts:
Flowers, leaves,
stems
Toxic plant chemicals:
unknown chemical
Animals/Human Poisoning:
Note: When an
animal is listed without additional information, the literature (as of 1993)
contained no detailed explanation.
Horses
Sheep
General symptoms of
poisoning:
Anorexia,
depression, incoordination.
Notes on poisoning:
Experiments on
sheep showed that animals ate the distasteful plant after a period of
starvation. Ingesting plant material equal to 3-4% of body weight produced
symptoms after 24 h. The primary symptoms were incoordination and listlessness.
Respiratory rates increased. Animals returned to normal within 36 h (Kingsbury
1964).
Swine
General symptoms of
poisoning:
Abdominal
pains, anorexia, depression, incoordination
Notes on poisoning:
In experiments,
swine ate the distaste plant material after a period of starvation. The animals
exhibited incoordination, dullness, some signs of abdominal pain, and aimless
wandering. The symptoms disappeared within 36 h. Symptoms appeared after
ingesting plant material equal to 3-4% of body weight. Symptoms could not be
reproduced a second time with further feedings (Kingsbury 1964).
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